I’ve noticed a shift in the literature in the past few years - amyloid beta is no longer the main point of interest, rather it’s tau. Do you think this re-evaluation of the biggest culprit of AD is promising? And, are BACE1 inhibitors really the “cure”?
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I’ve noticed a shift in the literature in the past few years - amyloid beta is no longer the main point of interest, rather it’s tau. Do you think this re-evaluation of the biggest culprit of AD is promising? And, are BACE1 inhibitors really the “cure”?
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